The role of folate transport and metabolism in neural tube defect risk

Mol Genet Metab. 1999 Jan;66(1):1-9. doi: 10.1006/mgme.1998.2787.

Abstract

Neural tube defects (NTDs) are common congenital malformations in humans. While etiologically heterogeneous, for the most part they are multifactorial in their pathogenesis, having both genetic and environmental factors contributing to their development. In recent years, there has been a great deal of epidemiologic evidence demonstrating that women who received multivitamins containing folic acid periconceptionally had significantly reduced occurrence and recurrence risks for producing infants with such malformations. Unfortunately, the mechanism(s) underlying the beneficial effects of folic acid is not well understood. In this article, we review the fundamental embryological processes involved in closing the neural tube, the relevant epidemiologic data on folic acid supplementation and relative NTD risk, as well as several recent studies of candidate genes for NTD sensitivity that are involved in folate transport and metabolism.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Biological Transport
  • Carrier Proteins / genetics
  • Carrier Proteins / metabolism
  • Folate Receptors, GPI-Anchored
  • Folic Acid / metabolism*
  • Folic Acid / pharmacokinetics
  • Humans
  • Neural Tube Defects / etiology
  • Neural Tube Defects / metabolism*
  • Neural Tube Defects / prevention & control
  • Receptors, Cell Surface*
  • Risk Factors

Substances

  • Carrier Proteins
  • Folate Receptors, GPI-Anchored
  • Receptors, Cell Surface
  • Folic Acid