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Original research
HnRNPR strongly represses splicing of a critical exon associated with spinal muscular atrophy through binding to an exonic AU-rich element

Authors

  • Tao Jiang Department of Neurology and Suzhou Clinical Research Center of Neurological Disease, Second Affiliated Hospital of Soochow University, Suzhou, Jiangsu, China Institute of Neuroscience, Soochow University, Suzhou, Jiangsu, China PubMed articlesGoogle scholar articles
  • Ruobing Qu Institute of Neuroscience, Soochow University, Suzhou, Jiangsu, China College of Chemistry Chemical Engineering and Materials Science, Soochow University, Suzhou, Jiangsu, China PubMed articlesGoogle scholar articles
  • Xuan Liu Jiangsu Key Laboratory for Molecular and Medical Biotechnology, Nanjing Normal University College of Life Sciences, Nanjing, Jiangsu, China PubMed articlesGoogle scholar articles
  • Yanjun Hou Jiangsu Key Laboratory for Molecular and Medical Biotechnology, Nanjing Normal University College of Life Sciences, Nanjing, Jiangsu, China PubMed articlesGoogle scholar articles
  • Li Wang Jiangsu Key Laboratory for Molecular and Medical Biotechnology, Nanjing Normal University College of Life Sciences, Nanjing, Jiangsu, China PubMed articlesGoogle scholar articles
  • Yimin Hua Jiangsu Key Laboratory for Molecular and Medical Biotechnology, Nanjing Normal University College of Life Sciences, Nanjing, Jiangsu, China PubMed articlesGoogle scholar articles
  1. Correspondence to Professor Yimin Hua, Jiangsu Key Laboratory for Molecular and Medical Biotechnology, Nanjing Normal University College of Life Sciences, Nanjing, Jiangsu, China; huay{at}njnu.edu.cn
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Citation

Jiang T, Qu R, Liu X, et al
HnRNPR strongly represses splicing of a critical exon associated with spinal muscular atrophy through binding to an exonic AU-rich element

Publication history

  • Received January 27, 2023
  • Accepted May 8, 2023
  • First published May 24, 2023.
Online issue publication 
October 24, 2023

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