A new point mutation involving a highly conserved leucine in the Btk SH2 domain in a family with X linked agammaglobulinaemia. | Journal of Medical Genetics

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Research Article

A new point mutation involving a highly conserved leucine in the Btk SH2 domain in a family with X linked agammaglobulinaemia.

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Cite this article as:: Ohashi Y, Tsuchiya S, Konno T

A new point mutation involving a highly conserved leucine in the Btk SH2 domain in a family with X linked agammaglobulinaemia.

Journal of Medical Genetics 1995;32:77-78.