Interleukin-6 and interleukin-1α production is associated with antigen-induced late nasal response☆
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2019, Journal of Affective DisordersCitation Excerpt :Atopic disorders are highly heritable (Willemsen et al., 2008; Strachan et al., 2001) and highly comorbid with each other (Ker and Hartert, 2009; Pinart et al., 2014), suggesting they may share or have overlapping genetic susceptibility (Willemsen et al., 2008; Fagnani et al., 2008; Nystad et al., 2005). In susceptible individuals, exposures that trigger an atopic response may result in elevated levels of inflammatory biomarkers such as interleukin-6 (IL-6) and C-reactive protein (CRP) (Wood et al., 2012; Ólafsdottir et al., 2005; Borish, 2003; Yokoyama et al., 1995; Gosset et al., 1993). Elevated levels of inflammatory markers have been correlated with a range of mental health outcomes (Howren et al., 2009; Dowlati et al., 2010; Raison et al., 2006; Miller et al., 2009).
Persistent nasal symptoms and mediator release after continuous pollen exposure in an environmental challenge chamber
2016, Annals of Allergy, Asthma and ImmunologyCitation Excerpt :Nasal symptoms are observed in patients with allergic rhinitis (AR) after nasal provocation with allergen-coated disks or nasal spray and comprise the immediate and late phases of AR.1–5
Allergic and Nonallergic Rhinitis
2014, Middleton's Allergy: Principles and Practice: Eighth EditionIL-1 and allergy
2010, Allergology InternationalAugmented Phl p 5-specific Th2 response after exposure of dendritic cells to allergen in complex with specific IgE compared to IgG1 and IgG4
2008, Clinical ImmunologyCitation Excerpt :The analysis demonstrated an upregulation of IL-6, VEGF, MCP-3 and IL-1α as a result of stimulation of MoDCs with Phl p 5/IgE-complexes in at least four out of five donors. In agreement with our result, these mediators have previously been shown to be upregulated in Th2 related diseases [30–34]. MCP-3 is an important eosinophil chemoattractant known to play a key role in the allergic response [30].
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Supported by INSERM (No. 90-06), by SERPA, and by grants from, Centre Hospitalier Universitaire de Lille (No. 93-02).